From the various known facts and indications, we can now come to an understanding of the likely cause of MG. The decisive experiment, in which antibodies from a MG patient attacked receptors in healthy muscles, shows that the basic problem is with the antibody production and not the muscle receptors of the myasthenics. This means that the muscle receptors are basically healthy and the antibodies are produced against something else and attack the muscle receptors only as innocent bystanders. The real target may actually be in the thymus itself, as it has been shown that the thymus contains muscle-type cells with acetylcholine receptors. As the thymus is obviously diseased, at least in all advanced cases of MG, this suggests that the antibodies may actually be formed against faulty receptors in the thymus itself. After all, the thymus develops antibodies against many other conditions, but does not normally become diseased itself as it does in MG. The conclusion is that thymus receptors become faulty and susceptible to attack due to manganese deficiency. Otherwise the autoimmune attack would not stop and patients would start rapidly improving within days of manganese supplementation.
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