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Vitamin E deficient chicks have been shown to develop serious abnormalities of the central nervous system. In other animals large amounts of '          old-age' pigment (lipofuscin) accumulates in the nerve cells. The development and function of all endocrine glands depends on it. The pituitary              gland has an exceptionally high content of vitamin E, 15 times higher than in other parts of the body, while in the adrenal glands it is almost 6                times higher.

Babies have a high requirement for vitamin K, otherwise the development of glands, nervous system and muscle structures will be weak or faulty. Breast milk has about 10 - 20 times more vitamin E than cows' milk and colostrum even much higher levels. It is similar with vitaminC and various other essential nutrients.

The importance of vitamin E in myasthenia gravis can be seen in a case report were the initial use of other vitamins improved the condition somewhat, but only after the addition of vitamin E did all symptoms of the disease disappear.

In a study using rabbits with experimentally induced myasthenia gravis more animals survived with high-dose vitamins B1, C and E than in the unsupplemented group. These same vitamins in mega-doses (very high amounts) were successfully used for myasthenia gravis patients as stated in several published reports. However, I found that on a high-quality diet only a moderate amount of vitamin-mineral supplements was required for permanent remission. A patient who recovered on a raw food diet with only minimal supplementation initially persisted with double vision but overcame this with a hot castor oil pack over the forehead and eyes.

While magnesium is an essential mineral and activates many enzymes, a large dose of a magnesium supplement acts as a muscle relaxant and             causes myasthenia gravis patients to deteriorate. Another problem is caused by the enzyme-poison fluoride. A well-controlled patient was               reported to wake up with extreme weakness one morning. It was later learned that the water supply had been fluoridated for the first time                 during the preceding day.

Another patient, after being symptom-free for years, developed extreme weakness on two separate occasions during a glucose tolerance test.             The same happened after a breakfast with much sugar.

Three patients in intensive care failed to respond to any myasthenia gravis drug treatment. However, they improved greatly on nutritional therapy. Whenever placebos were given instead of vitamins their conditions deteriorated again.

Manganese and the Thymus Gland

Manganese and the thymus gland are the keys to the development and treatment of myasthenia gravis. Numerous enzymes are activated by manganese and it is essential for the production of energy from glucose. It is equally important for the growth of bones, the development of the skeleton and the formation of cartilage. It is essential for the development and functioning of nerves and muscles, specifically it is involved with muscular contraction. When muscles are damaged, manganese leaches into the bloodstream and causes its level to rise.

Manganese deficiency causes defective growth, muscular weakness, lack of coordination and balance, reproductive abnormalities and disorders of the central nervous system. Manganese is required for a healthy immune system and it is also involved in the synthesis of acetylcholine.

While the thymus gland is best known for its importance in the development and functioning of the immune system, it has also other, less known functions which to some degree are similar to those of manganese.

The thymus is an endocrine gland situated behind the upper part of the breastbone. It increases in size until puberty and then gradually shrinks again. Severe stress, including infection, causes the thymus to shrink excessively and prematurely, especially if there are deficiencies of the anti-stress vitamins. The experimental removal of the thymus in animals resulted in a 60% reduction in the contractibility of muscles, while the capacity to work was reduced by 42%.

In MG, the thymus is generally abnormal, usually much enlarged (hyperplasia) and not infrequently containing tumors (thymomas). Administration of high doses of manganese reportedly causes the thymus to shrink to its normal size in a very short time and thymomas and symptoms of MG to disappear.

This manganese therapy for MG was discovered and tested in the 1940's and 50's in the U.S. by E. K. Josephson (A-albionic Research 1961).

The report of his first MG case with this new method is quite instructive. A 43-year-old female developed the symptoms of MG in 1932. She had intermittent X-ray treatments for thymoma over many years. Drug treatment was started later but gave only a slight transient improvement and after some months she failed to respond completely. Nutritional therapy was started in 1937 with high doses of vitamins A, B, and C, along with a high salt intake because of severe adrenal weakness, and glycine, an amino acid important for the muscles. Within three weeks the patient was much improved. The later substitution of part of the salt with potassium chloride caused acute glaucoma and had to be stopped.

After a year the therapy started to become ineffective and the condition deteriorated again. Now vitamin E was added in the form of wheat germ oil. The condition rapidly improved and symptoms of MG disappeared except for occasional mild relapses. However, after two years MG reappeared without relief from the treatment.

In 1942 manganese sulfate was added to the therapy. Within one week her muscle strength was better than at any time during previous treatments and all symptoms of MG disappeared. The thymus tumor that had previously been unsuccessfully treated with X-rays disappeared as well. Until her death ten years later from a heart attack she had no more symptoms of MG.

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