Guide to inflammation.
Low fat Plant diet lowers cholestrol more
Introduction to Dysautonomia
Dysautonomias include chronic fatigue syndrome (CFS,) syncope ( blackout), panic attacks, anxiety, tachycardia (IST,) irritable bowel syndrome (IBS,) postural orthostatic tachycardia syndrome (POTS,) and fibromyalgia type pain. Including "brain-Fog".
Sufferers of these conditions experience an imbalance, in the autonomic nervous system - an imbalance termed dysautonomia.
The autonomic nervous system controls the "unconscious" bodily functions, such as heart rate, digestion, and breathing patterns.
The autonomic nervous system consists of two parts: the sympathetic system and the parasympathetic system. The sympathetic system can
best be thought of as controlling the fight or flight reactions of the body, producing the rapid heart rates, increased breathing, and
increased blood flow to the muscles that are necessary when an individual is in danger or under stress.
The parasympathetic system controls the quiet body functions, for instance, the digestive system. In short, the sympathetic system gets the body ready for action, while the parasympathetic system gets the body ready for rest. In normal individuals, the parasympathetic and sympathetic components of the autonomic nervous systems are in perfect balance.
In people suffering from dysautonomia, the autonomic nervous system loses that balance, and at various times the parasympathetic or sympathetic systems dominate. Symptoms include frequent, vague but disturbing aches and pains, faintness (or even actual fainting spells), fatigue, anxiety attacks, tachycardia, hypotension, poor exercise tolerance, gastrointestinal symptoms such as irritable bowel syndrome, sweating, dizziness, blurred vision, numbness and tingling, anxiety and (quite understandably), depression.
Sufferers of dysautonomia can experience all the symptoms or just a few of them. They can experience one cluster of symptoms at one time, and another set of symptoms at other times. The symptoms are often unpredictable, but on the other hand they can be triggered by specific situations or actions. (Some people have symptoms with exertion, for instance, or when standing up, or after ingesting certain foods.) Since people with dysautonomia are normal in every way, when the doctor does a physical exam they often finds no abnormalities. Dysautonomia is part of small fiber neuropathy.
Obesity surgery appears to increase the risk for atypical Wernicke encephalopathy, a serious neurologic condition caused by thiamine deficiency confusion, ataxia, and nystagmus — that characterize Wernicke encephalopathy, many patients also presented with myriad atypical symptoms, including hearing loss, convulsions, peripheral neuropathy, and Korsakoff psychosis.
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No IVIg available is not a problem as their are better alternatives like Rituxan , Remicade. IgG is present in both IVIg and colostrum, one is expensive and the other is cheap.
" a normal EMG does not rule out CIDP or Small Fiber.
In the treatment of ITP, there may be multiple mechanisms of IVIG action. The platelet count increase occurring within several days of the initiation of therapy appears to be caused by diminished sequestration of autoantibody-sensitized platelets. This may be caused by interference with Fc receptors on the cells of the monocyte-macrophage system. A similar mechanism may operate in other autoimmune and alloimmune cytopenias. Sustained responses to IVIG may represent spontaneous remissions or may be related to an immunosuppressive effect of IVIG.
There are several possible mechanisms by which the infusion of large concentrations of immunoglobulins may have an immunosuppressive effect. The presence of IgG dimers in immunoglobulin preparations, a result of pooling samples from a large number of individual donors, likely represents the occurrence of idiotype-anti-idiotype complexes. There is evidence that anti-idiotype antibodies in IVIG react with epitopes on the autoantibodies in patients with thyroiditis or spontaneous factor VIII inhibitors. Alterations of T-cell subsets and of in vitro B cell function, both spontaneous and mitogen driven, have been reported in patients treated with IVIG. It is unknown if these observations are related to a mechanism of therapeutic effect.
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