God is our Guide   Number 1 diseases research foundations


CIDP info
 Anti-inflammatory Diet
Burning  Feet Home
Services Page
Chronic Fatigue
Autoimmune diseases
Bible healing
Celiac disease

Diabetic amyotrophy


 Guide to Chronic fatigue

Fatigue & Fibromyalgia, 


Fat guide

Autoimmune self attack



 Autoimmune & women

Autoimmune anemia

Autoimmune Ear

Autoimmune Thyroid

Autoimmune Fiber

Autoimmune Muscle

Small Fiber

Myasthenia Holistic

Skin hair nail spa

Memory clinic

Depression & anxiety

 addiction  & Drug Rehab

Parkinson Clinic

Epilepsy Clinic

Pain Clinic

Bone disorders clinic

Joint disorder clinic

Skin repair clinic

Neurology Clinic



 Heart disease & stroke 

Reduce weight

Prevent Osteoporosis

Some rheumatic disorders

 Information on  inflammatory  autoimmune diseases      

     For a complete simple guide on treatment of Diabetic Amyotrophy


    contact us 


               Diabetic Amyotrophy Similar to CIDP

What is Diabetic Amyotrophy?

Diabetic amyotrophy is a condition which occurs in patients with diabetes (more likely in those with type II than type I). It usually starts with a severe pain at night then involves weakness a muscles of the thigh, hip, and buttocks. The symptoms usually occur on one side of the body but may involve both sides. Untreated the condition can last five months. Recent studies suggest a role for immunomodulating agents in certain types of diabetic neuropathy, including diabetic amyotrophy. This condition is very similar to CIDP or GBS but affects mainly the proximal muscles. See the diagnosis page link is on left top of this page.

Who gets Diabetic Amyotrophy?

Older men and middle aged women  are more likely to acquire diabetic amyotrophy. Approximately 1% of those adults with diabetes will obtain this condition. Once it happens on one leg then it will affect the other.

How is Diabetic Amyotrophy Diagnosed?

This condition is diagnosed by history alone. There is elevated CRP and ESR.

How is Diabetic Amyotrophy Treated?

This condition frequently will improve with time and the body will heal itself. Physical therapy and strict observation of blood glucose levels are recommended to help diabetic amyotrophy. Medications may help with the pain. However the cidpusa e-book lists the simple protocol which will make the patient fully mobile by day five. The medicine ciprofloxcillin is used and in four days full recovery is achieved. IVIg helps diabetic amytrophy. Full protocol described in the e-book.Electronic stimulation is also effective treatment. Please see the electronictreatment guide. Theelectronic devices used.

Studies on diabetic polyneuropathy patients with IVIg

Pain Med. 2009 Nov;10(8):1476-80. Epub 2009 Sep 25.
Intravenous immunoglobulin for the treatment of diabetic lumbosacral radiculoplexus neuropathy.   Tamburin S, Zanette G.

Department of Neurological Sciences and Vision, Section of Rehabilitative Neurology, University of Verona, GB Rossi Hospital, Verona, Italy.

The objective of this study was to evaluate the effect of intravenous immunoglobulin (IVIg) therapy in diabetic lumbosacral radiculoplexus neuropathy (DLRPN) patients who did not respond to analgesic drug therapy and corticosteroids. Background. DLRPN is a rare painful condition that may occur in diabetes mellitus (DM). At the moment, there are limited therapeutic options for DLRPN.

We recruited five patients affected by type 2 DM and DLRPN. They were selected from a cohort of 13 consecutive DLRPN patients. Inclusion criteria were severe pain (visual analog scale [VAS] > 4/10) and no response to pain symptomatic therapy and corticosteroids.Patients were treated with IVIg (0.4 g/kg/day for 5 days). Outcome measures were VAS, time of onset and duration of pain relief, the Medical Research Council (MRC) scale for lower limb muscle strength, and walking distance. (EMG) were retested after IVIg.


Four of the patients had positive pain response after IVIg. VAS reduction started 5-10 days after IVIg infusion. Two patients underwent additional IVIg infusions due to pain reappearance after 7-18 months, again with positive response. VAS, MRC scale, and walking distance significantly improved at 1 month (Wilcoxon nonparametric test, two-tailed, P < 0.05). Electrodiagnostic testing was unchanged, but needle EMG showed reduction of denervation signs after IVIg.


IVIg may rapidly reduce pain and improve motor function in DLRPN despite previous negative response to corticosteroids. IVIg may be repeated in those patients who experience disease relapse. Future double-blind trials are needed to evaluate the role of IVIg in DLRPN

Pathalogy in CIDP and autoimmune diseases  World Wide Consultation by Internet