TGA

Transient Global Amnesia

Complete detail and explanation on Amnesia


Transient Global Amnesia

The cause of TGA  is like a epileptic fit. There is convincing evidence that external emotional stresses, such as sexual intercourse, immersion in cold water, or strenuous physical exertion, can trigger the associated loss of memory. TGA may be the result of a transient ischemic attack, a "mini-stroke." Transient ischemic attacks are caused by a temporary interruption of the blood flow to the brain.  Another possible cause of transient global amnesia is a basilar artery migraine, a type of migraine caused by the abnormal constriction and dilatation of vessel walls.

Patients suffering from transient global amnesia have undergone medical imaging techniques, for example magnetic resonance imaging (MRI) and positron emission topography (PT), in order to find out what biological changes cause a temporary lapse in memory. The symptoms of transient global amnesia seem to be the result of dysfunction in such regions of the brain as the diencephalon and medial temporal lobes.  The diencephalon is composed of the thalamus, epithalamus, subthalamus, and hypothalamus. The thalamus is associated with memory, and changes in its structure have been proven to result in amnesia.  Some MRIs have shown evidence of changes in the medial temporal lobes, indicating that patients had suffered from a transient ischemic attack. Nonetheless, many people that have undergone such tests have not shown any changes in the functioning of their brains.

These findings are in line with our neurobiological understanding of memory. Under normal functioning, there are three kinds of memory: working memory, declarative memory, and procedural memory. Working memory allows for short-term recollection, for example, it is responsible for your being able to remember the gist of the sentence you just read. It is associated with the temporary storage of verbal and visual information. The verbal working memory is localized to the frontal regions of the left hemisphere, while spatial working memory involves mainly the right hemispheres. Procedural memory is responsible for cognitive and motor skills, all learned, habitual actions, for example, my ability to type this paper without looking at the keyboard or my ability to ride a bicycle.  The anatomical basis for procedural memory appears to be the basil ganglia, thalamus, and the frontal lobes. Declarative memory, associated with the hippocampus, is all experiences and conscious memory, including people, events, objects, facts, figures, and names. The region of the brain termed the medial frontal lobe is particularly responsible for declarative memory function.

There is much evidence proving that damage to the medial frontal lobe, severely affects a person's ability to recall and form long-term memories. The most well-known clinical example involves a patient called H.M.  This pateint named H.M. was afflicted with epilepsy. Surgeons removed both of his medial temporal lobes in an attempt to cure him from his disease. However, in so doing, they profoundly damaged his memory. He could no longer form new memories, though all his memories from before the surgery were retained - in other words he had anterograde amnesia.  Therefore, it appears that the lack of functioning and blood supply of the medial temporal lobe produces the symptoms of transient global amnesia, and results in the inability to make and recall autobiographical memories.

Transient global amnesia fortunately has a very positive prognosis - its effects are never permanent and the episodes last for a relatively short period of time. However, the inability to remember can be extraordinarily frightening. It is a natural experiment because it shows fairly clearly that certain parts of the brain are involved with certain kinds of memory. We often see ourselves as unitary beings, but in fact we are made up of many different processes that make up who we are. Although much of the neurobiology associated with memory remains quite mysterious, transient global amnesia helps highlight the particular machinery of our personal narratives.

Current research at cidpusa has found that stress triggers a inflammatory response similar to PTSD and the inflammation can result in a spasm of particular blood vessels resulting in the loss of memory.

Some things have been said, yet so much more cannot be written about. See this message from God.

A study called Mr FIT was done and it showed people who took oral hypoglycemic drugs for ten years went on to also develop heart disease.  Allopathic drugs may trigger other diseases.



Page desribes sudden memory loss and treatment

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Memory Disorders

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To reduce stress the best vitamin is Niacin or Nicinamaide taken once a day. Please see more information on Niacin at Niacin page link

 

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Vitamin- D is veryimportant to reuce inflammation in any disease. To reuce inflmmation you must need to take vitamin -D. Here is a link to more info on Vitamin D.
You must also be on a anti-inflammatory diet. This will help reuce diseases and here is a diet link.

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